fat pad and visceral fat

[Pegasus]

Interesting discussion here. I know for me losing 20 lbs reduced my fat pad by between .5" and .75". I have had very little sugar this whole time.

Yes and sex is great for stress reduction.

 

[Dre7]

Interesting thread. Visceral fat is quite hard to get rid of for me. Any recent success?

 

[Physdoc]

The good news is that visceral fat is easier to reduce than subcutaneous (regular) fat because it metabolizes more readily (contrary to popular belief). You pubic fat pad is not visceral fat - it's subcutaneous fat. The bad news is how much visceral fat you're prone to get is highly dependent on things other that over-eating or being sedentary - like your genetics. The other bad news is that visceral fat, like regular fat, is a secreting organism, like glands. The difference is that for whatever reason, visceral fat secretes far more unhealthy compounds than regular fat. This is why visceral fat is linked to a host of diseases. Your best plan to reduce visceral fat is similar to shedding regular fat - exercise, diet, get sleep and think healthy. Stopping smoking can also help reduce visceral fat more than regular fat. Got bum genes and have more visceral fat - no worries, you just need to exercise and diet more and results will still come.

 

[Pegasus]

As stated in the thread the evidence is that cortisol has an effect on visceral fat and so does insulin imho .

This may be a missing link for some .

 

[Pegasus]

If anyone has followed the premise of this thread I invite comment , especially from those that have used the info.

 

[Pegasus]

Ok so have now read several studies suppoting the premise of this thread . Here is an example off Pub Med.

Quote

Int J Obes Relat Metab Disord

. 1998 Dec;22(12):1184-96.
doi: 10.1038/sj.ijo.0800745.
[h=1]The interactions between hypothalamic-pituitary-adrenal axis activity, testosterone, insulin-like growth factor I and abdominal obesity with metabolism and blood pressure in men[/h]R Rosmond 1, P Björntorp


Affiliations expand

• PMID: 9877254
• DOI: 10.1038/sj.ijo.0800745

[h=2]Abstract[/h]Objective: To examine potential interactions between abdominal obesity, endocrine, metabolic and hemodynamic perturbations.
Subjects: A subgroup of 284 men from a population sample of 1040 at the age of 51 y.
Measurements: Anthropometric measurements included body mass index (BMI, kg/m2), waist/hip circumference ratio (WHR) and abdominal sagittal diameter (D). Endocrine measurements were a modified, low dose (0.5 mg) dexamethasone suppression test (Dex), testosterone (T) and insulin-like growth factor I (IGF-I). Overnight fasting values of blood glucose, serum insulin, triglycerides, total, low and high density lipoprotein cholesterol, as well as resting heart rate and blood pressure were also determined.
Results: Arbitrary subdivisions of the men were performed to obtain subgroups of low T and IGF-I values (lowest decile, borderlines < or =13.13 nmol/I and < or =128.80 microg/l, respectively) and normal or blunted Dex. Significant relationships with BMI, WHR or D, and abnormal metabolic and hemodynamic factors, usually with the exception of total and low density lipoprotein cholesterol, were then found in subgroups with different endocrine profiles. These included men with a blunted Dex test with low T or IGF-I values, as well as men with a normal Dex test and low or normal T or IGF-I values. In addition, a group with isolated low Dex suppression, as well as another group without endocrine abnormalities, showed such relationships. These findings suggest that, in men, obesity factors are associated with metabolic and hemodynamic complications with or without the presence of perturbations of hypothalamic-pituitary-adrenal axis (HPA) regulation or low T or growth hormone secretion. In order to generate hypotheses concerning the nature of the impact of the endocrine perturbations in abdominal obesity and its metabolic complications, path analyses were performed, testing different models. These models included the endocrine measurements (Dex test, T and IGF-I), the WHR and D (representing abdominal distribution of fat), BMI (representing obesity), as well as insulin and triglyceride values (representing metabolic perturbations). The results showed a satisfactory fit (goodness-of-fit index: 0.945 - 1.0) for the path diagrams: Dex --> T/IGF-I --> WHR or D --> insulin --> triglycerides with additional direct input of blunted Dex on insulin values (see Figure 1). With BMI as determinant, essentially the same results were found with the addition of a direct pathway between Dex and BMI as well as between IGF-I-T and insulin (Figure 2). There was no evidence for pathways where WHR or BMI determined endocrine variables.
Conclusions: The results suggest that abdominal obesity with or without endocrine abnormalities exerts a major impact on abnormalities in metabolic and hemodynamic variables. Abdominal obesity seems to be dependent on endocrine abnormalities, which in turn show direct or indirect relationships to the metabolic and circulatory variables, including a direct pathway between HPA-axis perturbations and accumulation of total body fat as indicated by the BMI. It is therefore suggested that endocrine perturbations are followed by obesity and by storage of an elevated proportion of fat in visceral depots, followed by metabolic and hemodynamic abnormalities. This is statistical evidence which is supported by evidence of mechanistic links in previous studies, suggesting the possibility of causal relationships. The results also indicate subgroups of abdominal obesity and its associated metabolic and hemodynamic abnormalities, which might be due to the input of different pathogenetic factors.

 

[Pegasus]

So looking at this part .
Quote
It is therefore suggested that endocrine perturbations are followed by obesity and by storage of an elevated proportion of fat in visceral depots, followed by metabolic and hemodynamic abnormalities. This is statistical evidence which is supported by evidence of mechanistic links in previous studies, suggesting the possibility of causal relationships.
Unquote

Cortisol ,insulin and testosterone seem to be involved in both weight gain (or at least some weight gain) and in particular visceral fat .

 

[Pegasus]

Ok what has happened o me in recent times may be instructive .

So I got ill this caused amongst other things stress ,loss of muscle mass and I believe a crash in test levels .

Ok so we have increased cortisol and decreased testosterone . So I do dexa tests which give a readout not only of the total amount of fat but where it is in the body . So since last dexa my total amount of body fat remained the same but the amount of visceral fat went up . So fat distributed in a manner as suggested by this thread .

 

[bigbrokevo]

To be honest, what is Fatpad??? is that like extra fat you have in your lower abdomen???

 

[Pegasus]

Yes .
Now central deposited fat including visceral and fat pad appear related .

 

[Pegasus]

This is somewhat realated in that ft distribution did not follow the old fashioned model.

https://www.pegym.com/forums/exerci...-spot-reduction-*read-before-comment-*-2.html

 

[Pegasus]

Seeing increaseing evidence that low test is linked to visceral fat .

 

[Big Al]

Seeing increaseing evidence that low test is linked to visceral fat .

"Testosterone enhances catecholamine-induced lipolysis in vitro and reduces lipoprotein lipase activity and triglyceride uptake in human abdominal adipose tissue in vivo."

From: Lowered testosterone in male obesity: mechanisms, morbidity and management

 

[18x13goalreached]

obviously when your androgen's aren't dispersing of the nutrients correctly the capacity to hold fat gets much higher even at lower calorie diets this is common knowledge?

Why trt is crucial for hypogonadism

 

[Pegasus]

obviously when your androgen's aren't dispersing of the nutrients correctly the capacity to hold fat gets much higher even at lower calorie diets this is common knowledge?

Why trt is crucial for hypogonadism

There are 3 hormones involved in fat distribution as well the "capacity to hol fat " .

My thought on trt are well known .

 

[18x13goalreached]

trt is Australia requires stringent imbalances in your natural hormone production its a very healthy and great way to fix a medical issue?

There are also 5 androgen's

How many androgens are there?

The endocrine glands secrete 5 androgens through a similar pathway: testosterone, dehydroepiandrosterone sulfate (DHEAS), dehydroepiandrosterone (DHEA), androstenedione, and androstenediol, the latter of which has both androgenic and estrogenic activity

All of which effect fat gain and loss

That's not even getting into "hormones" so saying "their are 3" is simply incorrect.

 

[Pegasus]

trt is Australia requires stringent imbalances in your natural hormone production its a very healthy and great way to fix a medical issue?

There are also 5 androgen's

How many androgens are there?

The endocrine glands secrete 5 androgens through a similar pathway: testosterone, dehydroepiandrosterone sulfate (DHEAS), dehydroepiandrosterone (DHEA), androstenedione, and androstenediol, the latter of which has both androgenic and estrogenic activity

All of which effect fat gain and loss

That's not even getting into "hormones" so saying "their are 3" is simply incorrect.

Yet again read the damn thread .

 

[Pegasus]

I do not regard trt as "very healthy ". I regard it as a last ditch effort that might be done after repeated well thought out natural methods have failed . There is the tendency in some quarters to jump to it as an easy option

 

[18x13goalreached]

7.3 nmol and 170 ngol are my levels its not pleasant bro

More than happy to upload my hypgonadism diagnosis from my endocrinologist if you so require

 

[Pegasus]

7.3 nmol and 170 ngol are my levels its not pleasant bro

More than happy to upload my hypgonadism diagnosis from my endocrinologist if you so require

As stated trt might be medicaly indicated in which case I have no issue with it . I know low T causes a raft of unpleasant issues quite aside from being involved with visceral fat .